Probe Positioning & Scanning Technique
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Use a curved array transducer (e.g., 3–6 MHz) for general abdominal evaluation; for superficial zones (e.g., abdominal wall, anterior peritoneum), a linear probe (e.g., 7–12 MHz) may help detect subtle free air or echogenic peritoneal thickening.
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Typical patient position: supine, with possible slight left tilt or elevation of head-end if needed to optimize fluid or gas detection.
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Standard windows to examine include:
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Right upper quadrant (RUQ) — e.g., subcostal view between liver and kidney, to assess for fluid in Morrison’s pouch (hepatorenal recess).
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Left upper quadrant (LUQ) — splenorenal region.
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Pelvis — pouch of Douglas in women, rectovesical pouch in men for free fluid.
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Anterior abdominal wall or pre-hepatic space for free air (especially for suspected perforation).
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Use gentle sweeping and sliding to assess the full peritoneal cavity; in suspected peritonitis look for indirect signs (fluid, peritoneal thickening, fat stranding/gas) rather than always expecting the disease to appear as a “mass”.
📸 Key Sonographic Findings in Peritonitis
Here are common ultrasound findings suggestive of peritonitis:
✅ Normal Findings vs 🚨 Abnormal (Peritonitis) Comparisons
Normal Ultrasound:
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Minimal or no free fluid in the hepatorenal or splenorenal recesses.
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Peritoneal stripe appears thin, uniform, with no significant thickening or enhanced echogenicity.
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Mesenteric fat appears homogeneous, not hyperechoic or thickened.
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No abnormal gas under abdominal wall or suspicious comet-tail artefacts.
Findings suggestive of Peritonitis:
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Significant free fluid, often in dependent portions of the peritoneal cavity (e.g., Morrison’s pouch, pelvis).
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Fluid may be complex (echogenic), especially in infectious or hemorrhagic cases.
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Peritoneal thickening/enhancement, fat hyperechogenicity.
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Evidence of free air if perforation: linear echogenic interface, dynamic shifting.
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Possibly localized fluid collections/abscesses or bowel loop changes pointing to source.
🧠 Practical Tips & Key Considerations
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Clinical correlation is essential: Peritonitis is usually a clinical diagnosis reinforced by imaging findings (pain, peritoneal signs, fever, lab markers) rather than imaging alone.
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Ultrasound is operator-dependent; body habitus, bowel gas, patient cooperation affect image quality.
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Do not rely solely on absence of fluid to exclude peritonitis—small volumes of fluid or early disease may be missed.
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For suspected perforation, use anterior abdominal wall or sub-diaphragmatic windows to seek free air (ultrasound sensitivity is lower than CT but still useful).
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When fluid is highly echogenic (as in infected ascites/septic fluid) it may be mistaken for soft tissue—beware and scan thoroughly.
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Document images in at least two orthogonal planes, note location and character of fluid, any bowel/adnexal/organ abnormalities.
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In cases of suspected tubercular peritonitis the ultrasound may show ascites with fine septations, omental thickening, peritoneal nodules/strands

